To encourage correct use of the demonstrations in rats, monkeys and people of conditioned satiety and its mechanism, this paper briefly reviews publications that attribute control of meal size to learnt satiety.
Here, we report materials, device architectures, integration strategies, and in vivo demonstrations in rats of implantable, multifunctional silicon sensors for the brain, for which all of the constituent materials naturally resorb via hydrolysis and/or metabolic action9,10,11,12, eliminating the need for extraction.
Such an increase may be implied from the demonstration in adult rats that L5B and L6 cells are strongly and directly activated by the thalamus in vivo (Constantinople and Bruno 2013).
Although impaired short-term neurocognitive function has been recently demonstrated after CPB in rats, the demonstration of persistent long-term neurocognitive changes would be more relevant from a clinical perspective.
The novelty of our study is the demonstration in vivo in humans and rat models of a decrement of p21 in cystic kidneys as compared to non-cystic kidneys.
Stoltenberg, M. et al. Autometallographic demonstration of zinc ions in rat sperm cells.
This is consistent with results generated in rats [9,10] and humans [39] but contrasts with the demonstration that 11β-HSD1 in hamsters can inter-convert 7αOHC and 7-KC [40].
The tracer combination of the anterogradely transported Fast Blue and the retrogradely transported Nuclear Yellow allowed the demonstration that some phrenic nerve afferents terminate at the C4 and C5 spinal cord in rats (Revelette et al., 1988).
The demonstration that disrupting glutamatergic neurotransmission within a hippocampal-prefrontal system impairs object-in-place memory in rats, accords with these studies.
Functional isolation in rats.
