Spike-triggered averaging of the LFPO using the complex (Fig. 3C,D) or the simple (Fig. 3E,F) spike as trigger revealed the 15 Hz oscillation, demonstrating a tight phase-locking of the LFPO with both complex and simple spikes.
Concomitantly to the block of HIF-α accumulation, the exogenous expression of PHD2 makes unable ET-1 and ET-3 to increase VEGF protein levels demonstrating a tight link between PHD2/HIF-α PHD2/HIF-αependent VEGF expression (Figure 3F).
A visual appraisal of Fig. 2 can provide initial insight into candidate RG expression stability, as some RGs demonstrated a tight clustering of CT values (e.g. RG1, RG3, RG5, RG7, RG8, and RG9) while others demonstrated a spread in CT values (e.g. RG2, RG4, RG6, and RG10) across growth conditions.
Scanning electron microscopic examination of the nets demonstrated a tight association of individual cells with the fibers and nets examined after removal of cells showed no evidence that the growth of cells in any way changed the structure of the fibers.
Consistent with recent observations based on remotely sensed albedo, the AmeriFlux dataset demonstrated a tight linear relationship between canopy albedo and foliage nitrogen concentration (for forest vegetation: albedo=0.01+0.071%N, r²=0.91; forests, grassland, and maize: albedo=0.02+0.067%N, r²=0.80).
Research in recent decades has demonstrated a tight relationship between gesture and speech, with the two often "co-expressing" the same idea in two formats at once (McNeill, 1992).
Cross-correlation functions demonstrated a tight correlation between the unfiltered recorded signals along the frontal (0.81±0.02) and the sagittal (0.83±0.03) planes.
Previous results demonstrated a tight in vivo control of respiration by COX in skeletal muscle [24], indicating that p43 overexpression favours the use of oxidative substrates and a muscle fiber type switch.
In contrast to this finding, other reports using this same mutation in cell lines and in transgenic mice demonstrated a tight correlation between the anti-apoptotic and anti-proliferative functions of Bcl-2 and demonstrated these two functions could not be separated [28], [29].
Here we demonstrate a tight association between myocardial FA metabolic disorder and obesity-related cardiac remodeling.
The results demonstrate a tight and causal link between asynchronous GABA release and tonic inhibition.
