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It is, however, important to note that Weiskrantz et al's [1] classic study of DB demonstrates preserved visuomotor function in the presence of a visual delay.
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The borders demonstrated preserved inner retinal layers with atrophy of the outer retina and choroidal hyperreflectivity from transmission defect.
Immunofluorescence staining and flow cytometry analysis with the FITC-labeled MAb and scFv demonstrated preserved immunoreactivity by binding to CA125 antigen expressed on the membrane of NIH OVCAR-3 cells, aNIH OVCAR-3ng was seen with SKOV3 cells (Figure 2andB, C).
The radiolabeled IgG, F ab' 2 and Fab fragments all demonstrated preserved immunoreactivity.
Persons with action-blindsight report being "unaware" of visual stimuli within their impaired hemifield; however, such individuals demonstrate preserved saccades, visually guided pointing and tracking within their scotoma [see also 4].
Echocardiography demonstrated preserved ejection fraction and no atrial septal defect.
Electrophysiologic studies demonstrated preserved motor and sensory conductions in all four limbs.
Hematoxylin-Eosin (HE) staining demonstrated preserved follicular structures, having enlarged germinal centers in some Tet2 gt/gt mice.
This approach has been used to demonstrate preserved neurovascular coupling following cancer treatment [Zou et al., 2005].
Notably, Old + GLP-1 dogs demonstrated preserved coronary vasodilator response to hyperinsulinemia in the basal state, but not following the development of advanced DCM.
Despite demonstrating preserved cortical thickness relative to healthy controls at baseline, the PD-NC group subsequently developed significant frontal cortical thinning over the follow-up period.
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