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Infection of blood cells was demonstrated, virus was transmittable to indicator cells or to fresh PBMC and plasma samples from CFS patients were shown to contain virus-specific antibodies [7].
These strains consistently demonstrated virus antigen in the spinal cord white matter, often associated with inflammatory cells and parenchymal damage.
However, in contrast to these previous studies our study demonstrated virus gene expression and the formation of inclusion bodies, the latter being a characteristic of productive RSV infection.
Previous global phylogenetic studies of influenza have demonstrated virus mixing globally (3, 4, 11 ), a lack of interseasonal persistence in temperate regions (1, 2, 11, 12 ), and some evidence of persistence in subtropical regions (5, 13 ).
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Samples demonstrating virus were thawed and titrated on Vero cells as described above.
However, the majority of studies were unable to demonstrate virus multiplication and transmission to chimpanzees.
These findings are in agreement with our previous results demonstrating virus growth kinetics in HAEC.
This study demonstrates virus compartmentalization in the CNS and other body tissues in sheep presenting the neurological form of SRLV infection.
In this study, we were unable to demonstrate virus activity within mosquitoes collected despite sampling being done immediately before the short rainy season.
Here, a simple but efficient method is described for demonstrating viruses by negative staining, and its limit is discussed.
Detailed virus uptake analyses unambiguously demonstrated reduced virus uptake in various GL-treated cells.
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CEO of Professional Science Editing for Scientists @ prosciediting.com