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In a previous study [25], we demonstrated using Western blotting analysis that PRP stimulates VEGF receptor type 2 (VEGFR2) to accelerate endothelial cell motility and wound closure.
MCT1 expression was also demonstrated using Western blot analysis of proteins isolated from rat kidney (positive control tissue) and hypothalamus (Fig. 1C, lanes 1 2).
Bcl-xl and Mcl-1 reductions via siRNAs were demonstrated using western blotting.
Efficient knockdown of CYLD, TP53, FOXO3A, PAX6, NOTCH1, PTCH1 and HOXD10 is demonstrated using western blotting.
A highly elevated level of keratin 6a protein was also demonstrated using Western blot analysis.
As demonstrated using western blots, wild-type BRCA1 was able to induce a large reduction in endogenous IGF1R levels [ 20].
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Moderate reduction in protein levels is difficult to convincingly demonstrate using western blot.
The two distinct activities (biotin binding and anti-CA125) of the bfFp were demonstrated using ELISA, Western blot and confocal laser-scanning microscopy (CLSM).
Hippocampal α-CaMKII was significantly reduced following AAV-shCAM treatment as demonstrated using immunohistochemical and Western analysis.
This difference was demonstrated using three complementary techniques: Western blotting, quantitative RT-PCR, and immunofluorescence of cultured cells.
The nature of interactions between ERα and MS-KIF18A was demonstrated using recombinant and endogenous proteins by immunoprecipitation (IP) and western blot (WB) assays.
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