The relationship between the response and independent variables was demonstrated using a response surface plot.
The probabilistic implementation of explicit structural nonlinearity and foundation sliding in combination with the SSI effects are demonstrated using nonlinear response history analysis (RHA) of the structure with the foundation motions obtained from elastic SSI analyses, which are applied as input to fixed-base inelastic analyses.
In this study, the methodology is developed and demonstrated using the vibration response of a scaled aircraft wing simulated by a finite element (FE) model.
The proposed concept was successfully demonstrated using a developed C V response tunable MEMS varactor and a monolithically-integrated spiral inductor.
This response was demonstrated using UV-treated virus particles [3], [5], [56], [57], cycloheximide [4], [6], glycoprotein-deficient mutants [4], [5] or replication-deficient amplicon vectors [55].
We have previously demonstrated, using this model, a similar response of an additive effect of burn and disuse on muscle function and bone health [ 30, 31].
In contrast, the induction of antigen-specific Th1 immune responses was demonstrated using an infection model of mice with Leishmania major [ 22, 54].
Next, the utility of the internal RTD in the interpretation of tracer response curves is demonstrated using both theoretical mathematical models and measured tracer results in maldistributed flow fields.
Despite the clear role of p53 in the apoptotic response to oxaliplatin demonstrated using the HCT116 isogenic system that differs only in the presence or absence of a functional p53 protein, the mutational status of this tumour suppressor gene could not predict the apoptotic response to 10 μ M oxaliplatin, suggesting that additional factors modulate sensitivity to this agent.
The contribution of the Saps to mucosal and systemic infections and their involvement in adherence, tissue damage and evasion of host immune responses was clearly demonstrated using SAP-deficient mutants and protease inhibitors [25].
