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These results demonstrated that induction of IL-6 occurred somewhere between 100 fg/mL and 1 pg/mL.
Our earlier studies demonstrated that induction of I/R in the intact murine heart resulted in significant upregulation of RAGE, particularly in cardiomyocytes.
Prior studies have demonstrated that induction of IL-1β processing through activation of the NLRP3-inflammasome in response to the pore-forming toxin Nigericin requires intracellular potassium depletion.
Data was generated that demonstrated that induction of mitochondrial swelling along with enhanced Hsp70 and 90 expression could be reproduced in another rat glioma cell line, F98.
A recent report also demonstrated that induction of agr expression leading to increased production of multiple proteases may also serve as a specific means of dispersal from an established biofilm [40].
We have further demonstrated that induction of IFNβ, the first type I IFN to be produced in response to viral infections, reduces SIV replication in vitro in primary macaque macrophages by a transcriptional mechanism.
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We were, however, not capable of demonstrating that induction of more apoptosis or more necrosis results in differences in transepithelial passage of antibiotics.
To demonstrate that induction of caspase 2 and 3 activities were specific, we treated MON cells in the presence and absence of p53 and also in the presence and absence of specific caspase 2 or 3 inhibitors.
The results of the study demonstrate that induction of OHC apoptosis after the noise exposure is an extremely rapid process.
We demonstrate that induction of a specific subset of cytokines is dependent upon integrin α3β1-mediated endocytosis of the ligand.
Our previous study demonstrates that induction of NKG2D ligands in the WSX1-engineered TC1 and AT84 tumor cells are associated with tumor growth inhibition [12].
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