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Several studies, however, have demonstrated severe phenotypic and functional impairment of DCs in patients with breast cancer [ 11, 12].
Transthoracic echocardiography demonstrated severe AS.
An echocardiogram on hospital day 2 demonstrated severe biventricular dysfunction.
Follow-up CT scan demonstrated severe mediastinal contamination and left-sided loculated pleural empyema (Figure 2).
A brain computed tomography (CT) scan demonstrated severe pneumocephalus (Fig. 2).
The images demonstrated severe dilation of the ventricles and sulci suggestive of cerebral and cerebellar cortical atrophy.
Endoscopic retrograde cholangiopancreatography (ERCP) demonstrated severe intrahepatic bile duct dilatation and a filling defect in the mid-CBD.
Ischemic control animals demonstrated severe deterioration of renal function, renal morphology and a significant renal oxidative stress.
Following return of spontaneous heart rhythm and blood pressure, a complete TEE exam demonstrated severe aortic stenosis with an aortic valve area on 0.8 cm2.
Mutant mice with DMP1 point mutations changing S89 to glycine (S89G), which completely eradicated glycosylation of the protein, demonstrated severe BBB disruption.
The NSS demonstrated severe motor deficits at 3 h after MCAO in both groups (BMSC group, 9.90 ± 0.88; vehicle group, 10.20 ± 1.03).
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