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If Glimcher's results could be demonstrated in human brains, they might undermine a lot of neuroeconomics, and many in the field tend to downplay his work.
Practicality of the sensor was demonstrated in human urine samples.
Yet, the efficiency and specificity of base editors has not been demonstrated in human embryos.
However, these approaches have only been demonstrated in human tumor-derived cell lines or mouse models harboring mtDNA mutations.
Yet, increased CB1R levels were demonstrated in human PCa specimens compared to those of the benign samples [10].
Sensitization of Ha-ras-related cells to 5-FU was also demonstrated in human bladder cancer cells.
Moreover, recent epidemiological studies have demonstrated in human the incidence of dioxin exposure on enamel hypomineralisation and hypodontia.
Furthermore, practical feasibility of the sensor has been demonstrated in human serum and contact lens cleaning solution to determine glucose and H2O2, respectively.
Ligand density dependent cell substrate interactions were demonstrated in human foreskin fibroblasts, human umbilical vein endothelial cells, and rat neural stem cells.
We previously demonstrated in human epidermoid carcinoma KB cells that UVB irradiation activates PPARγ via the generation of multiple oxidized glycerophosphocholine species with PPARγ ligand activity.
The question of whether DHA regulates the expression of SAA to affect lipid metabolism and increase lipolysis needs to be demonstrated in human adipocytes.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com