Analysis by eosin and haematoxylin (H&E) staining demonstrated damaged but intact cells with intact nuclei in the urinary sediment.
Although this was not the focus of the present study, a previous study demonstrated that damage to type IV collagen exposes cryptic sites that alter interactions with endothelial cells (Xu et al, 2001).
Optical manipulation demonstrated that damage to the embryo initial cell induces cell fate conversion of the suspensor cell to compensate for the disrupted embryo initial cell even after cell fate is specified.
It has been demonstrated that damage to the pedal ganglion of the Pulmonate Melampus results in locomotor deficits that recover spontaneously following a recovery period.
Groups received STZ, demonstrated cellular damage to the pancreatic acini and islets, which showed pancreatic β-cell damage and degeneration with asymmetrical vacuoles.
Studies of RA synovial fluid and tissue have demonstrated oxidative damage to hyaluronic acid [ 29], lipid peroxidation products [ 30, 31], oxidized low-density-lipid proteins (LDL) [ 32], and increased carbonyl groups reflective of oxidation damage to proteins [ 32, 33].
Simulated lesions demonstrated that damage to the layers of the dorsal stream had a greater impact on repetition accuracy, whereas damage to layers of the ventral stream had a stronger impact on comprehension accuracy.
Wiesner et al., on the other hand, demonstrated that damage to a different type of biomolecule, mitochondrial DNA (mtDNA), not only accumulates during aging and in age-related pathologies, but may be enhanced by the turnover and metabolism of well-known signaling molecules, catecholamines.
(3) High serum levels of anti-CCP-2 Abs have been associated with an erosive disease outcome in RA. (4) RA synovial fluid and tissue have also demonstrated oxidative damage to hyaluronic acid, low-density-lipoproteins (LDL), proteins, cartilage, extracellular collagen, and intra-cellular DNA.
