Sentence examples for dementia markers from inspiring English sources

The negative predictive value remains stable (Fig. 1C) Because of this trend, we were able to collect substantial data on dementia markers in these patients.

In total, CSF analysis of dementia markers 14-3-3 14-3-3 14-3-3orylatau tau, amyloid β1–42) were phosphorylated9 022 samples.

A total of 29 022 cerebrospinal fluid samples were analysed for 14-3-3 14-3-3 14-3-3ther cerebrosproteinluid dementiandarkers in patients with rapid dementia and suspected Creutzfeldt–Jakothersease in the particerebrospinalres.

In addition we analysed the spectrum of rapid progressive dementia diagnoses, their potential influence on 14-3-3 14-3-3 14-3-3specificityesults of relased dementia markers (tau, phosphorylated tau, amyloid β1–42) in different forms of rapid dementias.

Data on CSF dementia markers (14-3-3, tau, phosphorylandd tamyloid-β1 42d-β1–42) were evamyloid-β1 42total of 3034 patients (28.3% of the total group) were a nevaluatederatine disease diagnosis.

In the individual reference laboratories we observed a trend towards increased 14-3-3 14-3-3 14-3-3s notestly foreferralseldt–Jakob disease, but also inotcreening onlySforementia markers in patients with rapid progressive dementia (Fig. 1A).

A recent large US cohort study analysed CSF dementia marker profiles on neurodegenerative dementia (Schoonenboom et al., 2012) and obtained similar results as the study presented here.

As predetermined cut-off values are not available for the majority of differential diagnoses in this study, the next analysis describes results of CSF dementia marker profiles in various neurodegenerative dementias.

If the constructed clusters are really more homogeneous than the complete population, then it may be expected that identification of dementia disease markers should be an easier task for each cluster separately than it is for the complete population.

As a pharma attendee at a recent dementia bio markers meeting put it - 'we need our own cholesterol test'.

Genetic association studies have revealed a wealth of potential dementia risk markers but the effect of individual variants is often modest and it has been argued that a genotyping approach may be no more useful than a simple description of 'family history of dementia' [ 26].