Sentence examples for deleterious interactions from inspiring English sources

The study of the deleterious interactions between an agent and a biological system also falls within the scope of toxicology.

These results clearly demonstrated that deleterious interactions in Group III (interactions of Sa and Sb pollen sterility loci) have more pronounced effects on the pollen sterility of polyploid rice compared to other groups.

In the context of hybrid PET/MRI, conventional PMT detectors can only be used if the PET unit is placed sufficiently far from the MRI unit to virtually eliminate deleterious interactions, thus leading to a sequential system design [4].

Semen contains high concentrations of soluble proteins that could protect sperm from potentially deleterious interactions with virus capsids.

Hybrid dysfunctions, such as sterility or inviability, result from failed (or novel deleterious) interactions between the genomes of the two parent species.

Deleterious interactions between such rare alleles in compound heterozygous carriers have the potential to account for a substantial public health burden.

For an alternative binding interaction to be deleterious, the proteins need to have a deleterious interaction (as defined by biological fitness) and to have the potential to interact at the same time, in the same cellular location, at the right concentration [ 42].

Understanding the key mechanisms underlying this deleterious interaction may provide opportunities for the design of effective therapeutic strategies.

However, if the deleterious interaction between the quantum register and its environment is subject to correlations between qubits and non-Markovian effects, more care must be exercised in the assessment of how errors accumulate, and how they may be corrected [36, 37].

Thus, targeting p75NTR could protect neurons from Aβ by at least three possible mechanisms: i) blocking a deleterious interaction between Aβ and p75NTR; ii) down-regulating deleterious signaling (calpain, GSK3β and c-Jun) which mediates Aβ toxicity; iii) upregulating survival signaling (AKT) which is normally inhibited by Aβ and which can antagonize Aβ mechanisms.

This suggests that the deleterious interaction between RA fibroblasts and cartilage is mediated by CD44 on the fibroblast cell surface.