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In the current study, we looked specifically at (1) whether hands-on experiences affect performance (Experiments 1 and 2) and (2) whether the effects persist after a delay (Experiment 3).
Therefore, we delivered fractionated radiation in five daily fractions of 2 Gy (XRT1 5) to PSN-1 tumors (Growth delay experiment II; Figures 4d and e).
We next tested the efficacy of VE-822 in a different tumor xenograft, MiaPaCa-2 (Growth delay experiment III; Figures 4f and g).
We then asked whether mice bearing PSN-1 xenografts would show a better response to radiotherapy when co-treated with VE-822 (Growth delay experiment I; Figures 4b and c).
In growth delay experiment V, a significantly reduced vessel density was observed after therapy with gemcitabine, whereas addition of VE-822 and/or XRT to gemcitabine did not alter vessel density significantly.
For the growth delay experiment (n=7 per group), the virus was administered when the tumors reached 180 200 mm in size, and twice thereafter at 4-day intervals.
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time, that is, the delay experimented by video packets in the transit from source to sink.
Kinetic modeling with a detailed mechanism showed that predicted ignition delay times are in good agreement with shock tube and rapid compression machine ignition delay experiments.
Full results of the diagnosis delay experiments with the declining AVEi assumption are given in Support Information Text S1.
Tumour xenograft growth delay experiments were also performed.
Two separate delay experiments (1 hr and 2 hr) were performed.
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