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The equivalent fatigue limit expressed by the applied criterion is compared with the crack size dependent fatigue limit described by El-Haddad correction in order to define a defect size acceptability criterion.
Others define a defect as critical sized if defect does not heal spontaneously within the lifetime of the animal or experiment [ 10– 13].
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Following previous studies performed under (mostly) static or time-harmonic conditions, the TS field is here considered as a natural and computationally efficient approach for defining a defect indicator function.
The aim of this study is to understand the impact of periodic surface micro-geometry patterns (obtained by High Speed Machining process) on the fatigue behavior of the AA7050 aluminium alloy and to define a proper defect acceptability criterion.
This study was designed to define a critical size osteochondral defect in the knee of rats and to investigate a possible association between osteochondral defects and degeneration of the surrounding joint surface.
Finally, fatigue life predictions have been compared with the real experimental results from the S N curves of the samples, in order to define an equivalent initiation defect due to processing conditions, which can be used in the design or integrity assessment of any component.
Although we cannot exclude an effect of referral bias, these observations merit further studies addressing the question whether age, gender and clinical features define a risk profile for molecular defects in AAA patients.
A wound is defined as a defect or break in the skin, formed due to physicochemical or thermal damage or as a result of a pathological condition.
In the ASTM Standard Guide for Preclinical in vivo Evaluation in Critical Sized Segmental Bone Defects (F2721-09) CSD model is defined as "a defect that will not heal without intervention" [ 9].
Peptic ulcer disease was defined as a defect in the gastric or duodenal wall that extended through the muscularis mucosae into the deeper layers of the wall (submucosa or the muscularis propria).
A cerebral infarct was defined as a defect of the brain parenchyma with a signal intensity that is isointense to that of cerebrospinal fluid on all pulse sequences (i.e., FLAIR and proton density/T2-weighted) and that has a minimal diameter of 4 mm, with the exception of infarcts in the cerebellum, which had no size criteria.
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