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No vascular or neurological deficits were present.
Similar deficits were present in most neglect patients in a newly developed space bisection task.
After PVI, regional deficits were present in ten patients (62.5%) with newly emerging deficits localized in the inferolateral wall.
Prominent contralateral deficits were present in sensorimotor response latency and contralateral circling was induced by apomorphine challenge (both P < 0.05).
Although P50 deficits were present in patients, there was no evidence for an association between COMT Val108/158Met polymorphism and the P50 biomarker.
After PVI, regional deficits were present in two patients in the early but in the ten patients (62.5%) in the late images including the four patients with preexisting deficits which remained unchanged.
Similar(37)
In cauda equina syndrome cases, sphincter deficit and sensory deficits are present in addition to motor deficits of the lower extremities.
However, while unilateral deficits are present in patients after ACLR, these may not be evident during activities involving both lower extremities.
In conclusion, deficits are present in some but not all areas of cognitive functioning during the early stages of bipolar I disorder.
Therefore, while unilateral deficits are present in patients after ACLR, these may not be evident during activities involving both lower extremities.
Cognitive deficits are present in a large majority of Bipolar Disorder (BD) patients and known to be a marker of bad prognosis.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com