Sentence examples for deficient red from inspiring English sources

G6PD deficient red cells (G− RBCs) display a failure of the protective response to oxidant stress, which leads to irreversible oxidation of glutathione [1], [2], [4] [6].

New findings concerning the mechanisms of G6PD deficient red cell destruction may facilitate our understanding of the large individual variations in susceptibility to pro-oxidant compounds and aid the prediction of the hemolytic activity of new drugs.

Our results show that treatment of G6PD deficient red cells with diamide (0.25 mM) or divicine (0.5 mM) causes: (1) an increase in the oxidation and tyrosine phosphorylation of AE1; (2) progressive recruitment of phosphorylated AE1 in large membrane complexes which also contain hemichromes; (3) parallel red cell lysis and a massive release of vesicles containing hemichromes.

We think this statement is valid because treatment with eculizimab increases the level of PIGA deficient red cells.

") seem to suggest that the relative expansion of the "PNH (and concomitantly G6PD deficient) red cells" following eculizumab treatment could have played a role in the co-trimoxazole associated hemolytic attack.

Since it has been demonstrated that treatment with eculizumab changes, the relative proportion of GPI-AP deficient red cells [ 19], patients who received eculizumab treatment were censored at start of this treatment.

Plant fresh weights and leaf SPAD values of Fe-sufficient (green) and Fe-deficient (red) plants were measured at 0 and 7 days after the Fe deficiency treatment.

Immune system scavengers called macrophages seem to be the predators: If the spleen, headquarters to the macrophages, is removed, the CD47-deficient red blood cells were spared.

Root concentrations of a NA (n = 5), b DMA (n = 5) and c DMA secretion (n = 3) from Fe-sufficient (green) and Fe-deficient (red) rice roots were quantified by LC-MS.

Expression of a OsNAS1, b OsNAS2, c OsNAS3, d OsNAAT1, e OsDMAS1, f OsTOM1 and g OsYSL15 genes in the roots of Fe-sufficient (green) and Fe-deficient (red) rice plants.

Specific syndromes of disease, i.e. enzootic ataxia and osteochondrosis, that have previously been reported in Cu-deficient red deer herds in Norway [ 9, 15], were not observed.