Sentence examples for deficient fish from inspiring English sources

After 17 weeks the deficient fish had lower α-T concentrations in viscera, lower whole-body crude protein, total lipid, dry matter, hematocrit, hemoglobin, lymphocyte, alternative complement activity (ACH50), and survival after exposure to stressful water temperatures (36 37 °C) than those fed the α-T-supplemented diets.

Together, these data indicate that p53 is able to rescue the survival of tert-deficient fish but not telomere length.

We set out to investigate whether the mbnl2-deficient fish showed any muscle alteration parallel to those observed in DM.

NEMO was able to rescue in a dose-dependent manner the wild-type vascular phenotype in TNFRSF1B-deficient fish (Fig. 2C).

Furthermore, we assessed the effect of the permanent absence of p53 in tert-deficient fish by obtaining the G1 of double-mutant tert −/− ; p53 −/− zebrafish.

Because caspase-8 is the main initiator caspase involved in TNFR signaling, we analyzed caspase-8 activity in TNFRSF1B-deficient fish.

DOI: http://dx.doi.org/10.7554/eLife.06545.019 We show that Tjp1a-deficient fish develop multiple interruptions of the dark stripes in the trunk by light stripe structures composed of dense iridophores covered by compact xanthophores.

As shown by Anchelin and colleagues, telomerase-deficient fish showed a p53-dependant premature aging and reduced lifespan in the first generation, as occurs in humans but not in mice (5).

Interestingly, one of the chemicals, aminophylline, a nonselective phosphodiesterase (PDE) inhibitor, demonstrated a promising capacity to restore normal muscle structure and upregulate the cAMP-dependent protein kinase A (PKA) pathway in dystrophin-deficient fish (Kawahara et al., 2011).

The p53-deficient fishes were originally generated by TILLING.

In the zebrafish, defective mpv17 altered only skin pigmentation and normal viability of the mpv17-deficient fishes was suggested to result from the functional redundance of the mpv17 paralogues (Krauss et al., 2013).