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This failed to rescue normal feeding of the pups, suggesting that lack of oxytocin was not the cause of the deficient feeding.
A potentially deficient feeding response might in turn be due to underlying neurological defects, as suggested by examination of other genes expressed in the central nervous system [37], [38].
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Folate-deficient feeding significantly enhanced mtDNA content and overall abundance of the D-1 mtDNA deletion in brain of Ung−/−, but not of wild-type mice.
Also, Table 2 shows the increased level of HA induced by vitamin B6-deficient feeding.
We have found that traditional iron-based catalysts for the Fischer Tropsch synthesis can be successfully used to obtain high added-value products from CO2 even in the presence of H2 deficient feed streams.
Despite the significant alterations in fat mass and hepatic triglyceride levels in female hypothalamic NPY neuron-specific Y2 deficient mice, feeding behaviour and body weight were not altered in male or female knockouts, although significantly increased NPY and decreased POMC mRNA levels in the Arc would have been predicted to induce hyperphagia and weight gain.
Our results were obtained by vitamin B6-deficient feeding to mice, which induced the vitamin B6-deficient burden on 3xTg-AD mice.
To this end, we compared the extent of steatohepatitis in wild type and TLR-2 deficient mice fed a methionine and choline deficient diet (MCDD) enriched with polyunsaturated corn oil or coconut oil as the saturated fat.
Six-month-old male apoE deficient mice fed a normal chow underwent ligation of the common left carotid artery, and were randomly assigned to receive either vehicle or angiotensin II (Ang II, 1.4 mg/kg daily) via a subcutaneously implanted osmotic infusion pump.
The initial report on CD36 deficient mice fed a chow diet with 4% fat described a modest increase (30%) of plasma cholesterol and HDL as compared to WT mice[11] but increased HDL were not noted in other studies[5], [29].
Indeed, a recent report demonstrated that high serum cholesterol levels in apolipoprotein E (ApoE) deficient mice fed on a HC diet, resulted in high susceptibility of to Mtb [28], although ApoE appeared to have a more pronounced influence on the regulation of plasma cholesterol than SR-B1.
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