MMP2 and MMP14 deficient mice display diminished ductal elongation, whereas MMP9 deficiency has no effect [ 13].
Thus, CCR2-deficient, and ACKR2-deficient, mice display reciprocal dermal lymphatic vessel density phenotypes.
In addition, like p50-deficient mice, BCL-3-deficient mice display reduced Peyer´s Patches but not a complete absence of them as seen in p52/p100-deficient mice [ 53].
Moreover, GCK-1 deficient animals display a significant increase in germ cell death.
AID deficient mice display a mild B cell hyperplasia and enlarged germinal centers [6].
Likewise, Thy-1 deficient mice display more severe lung fibrosis in response to an inflammatory injury than wildtype littermates.
In analogy with the latter result, resting granulocytes from Nox2 deficient patients display normal IHv density [1].
Indeed, Batf-/ deficient mice display normal Th1, and Th2 response but are deficient in Th17 differentiation, and are resistant to experimental autoimmune encephalitis[63].
Chgb deficient mice display reduced levels of catecholamines released per quanta [ 41].
Malonyl CoA Decarboxylase Deficient Mice Display Minimal Infarct During In Vivo Ischemia/Reperfusion Wei Wang, John Edward R. Ussher, Shaohua Wang, Jason R.B. Dyck, Gary D. Lopaschuk Cardiovascular Research Group.
