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OsHORZ1 expression shows only slight induction in response to Fe deficiency (Table 1) (Kobayashi et al. [2013]).
The transcript levels of the OsIRT1, OsIRT2, and OsNRAMP1, but not OsNRAMP5, are strongly upregulated under conditions of Fe deficiency (Table 1).
We compared the motifs generated by MAMA, MEME, MotifSampler and SIFT from the top 50 rice genes upregulated by Fe deficiency (Table 1, 2, Additional file 1).
The expression of OsYSL2 and ENA1 are strongly induced under conditions of Fe deficiency (Table 1) (Koike et al. [2004]; Lee et al. [2009]; Ishimaru et al. [2010]; Nozoye et al. [2011]; Ogo et al. [2014]).
Numerous human gastrointestinal disorders, such as celiac disease, are also associated with villous atrophy and diarrhea and may represent secondary or acquired forms of sucrase-isomaltase deficiency (Table 1) [21].
Although OsVIT1, OsVIT2, and MIT expression are repressed under conditions of Fe deficiency (Table 1) (Bashir et al. [2011b]; Zhang et al. [2012]), the contribution of these transporters to root Fe flux is not well understood.
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Leaf miR408 was down-regulated by B-deficiency (Table 2), as previously reported on N-deficient seedlings of Arabidopsis [ 27].
However, SPL9 in C. sinensis roots was down-regulated by B-deficiency (Table 3).
However, root level of pyruvate dehydrogenase E1 α subunit (R83) increased in response to Mg-deficiency (Table 2).
Leaf miR477 was up-regulated by B-deficiency (Table 2), as previously reported on salt-stressed Populus cathayana plantlets [ 56].
However, the level of late embryogenesis abundant (LEA) protein (L116) in leaves decreased in response to Mg-deficiency (Table 1).
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