TGF-β enables the tumor to efficiently evade immune surveillance by potently suppressing the maturation and function of DCs, which leads to a deficiency in antigen presentation and immune response (Grauer et al., 2007).
It gives an overview of techniques to overcome potential deficiencies in result presentation, using method effectiveness models such as 'Per Protocol' (PP) or 'As-Treated' (AT) that may give more accurate clinical meaning to the presentation of obstetrical results.
(We should remark that some authors also consider − | x | + | r | and call this the deficiency of the presentation P.) Remark 1 In both group and monoid cases, if the presentation P in (1) is efficient or inefficient while it is minimal, then it always has a minimal number of generators.
It is thus likely that the previously reported cross-presentation deficiency in ΔY-derived DCs resulted from the inability of these tyrosine-mutated surface Kb molecules to traffic through endocytic loading compartments (ELCs) [21].
In this context, we propose that the cross-presentation deficiency in ΔY mice results in T cells receiving inadequate activation signals which do not clonally expand and instead undergo apoptosis with the manifestation of a large reduction in CD8 T cell number at the peak of Listeria infection response.
Three variables were selected in multivariate logistic regression to predict severe ADAMTS13 deficiency at clinical presentation (Table 4): creatinine level, platelet count, and presence of ANA.
We have previously described the adult presentation of H6PDH deficiency in three females (4).
We developed an algorithm to predict severe ADAMTS13 deficiency at clinical presentation.
It doesn't quite make up, however, for the deficiencies of the mysteries, which are convoluted in conception and prosaic in presentation.
This observation demonstrates that the SMC inability in presentation cannot lie in a defect in the antigen processing; thus further investigations will be necessary to understand the mechanisms underlining this deficiency.
To support this conceptual view, our group recently reported for the first time, in a series of 13 patients with MDR3 deficiency, imaging presentations mimicking sclerosing cholangitis in two patients at MR imaging [30] (Figs. 8 and 9).
