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There are other types of trioctahedral smectites in which the net charge deficiency arises largely from the imbalanced charge due to ionic substitution or a small number of cation vacancies in the octahedral sheets or both conditions.
Nutritional iron deficiency arises when physiological requirements cannot be met by iron absorption from diet.
This deficiency arises because most research on the production of high-titer phage suspensions was completed over half a century ago and more recent advances on these methods have not been compiled in a single publication.
In contrast, type 2 diabetes (T2D) is typically a disease of adulthood (although more cases are now being seen earlier in life), where a relative insulin deficiency arises due to insulin resistance and abnormal pancreatic β cell function [1].
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Besides, the nonlinear oscillation of earthquake can be modeled with fractional derivatives [8], and the fluid-dynamic traffic model with fractional derivatives can eliminate the deficiency arising from the assumption of continuum traffic flow [9].
However, the present study further shows that the deficiency arising from small MSAs can be partially offset by the shuffling algorithm (Fig. 4).
In conclusion, these results indicate that phenotypically heterogeneic and penetrant cardiac malformations in Cited2 deficiency arise from a primary requirement in epiblast derivatives for left right patterning with a secondary cell-autonomous role in the mesoderm for septation.
The F1 aneuploid monosomic for chromosome 26 was unusual in that the deficiency arose de novo in 3-79 pollen, i.e. not via transmission from the maternal TM-1−like stock.
These results indicate that the penetrant and phenotypically heterogeneic cardiac malformations in Cited2 deficiency arise from a primary requirement in epiblast derivatives for left right patterning with a secondary cell-autonomous role in the mesoderm for septation.
These results indicate that phenotypically heterogenous and penetrant cardiac malformations in Cited2 deficiency arise from a primary requirement in epiblast derivatives for left right patterning, with a secondary cell-autonomous role in the mesoderm.
In the current study, the high overall CNV burden in HNPCC/LS patients could be due to their MMR deficiency arising from mutations in MMR genes, supporting the idea that deficiency of MMR occurs first and the adenoma evolves from the MMR-deficient cell [ 65].
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