X-ray photoelectron spectroscopy data indicate that Sr deficiency changes valence states of B-site cations.
To clarify the onset of Fe deficiency, changes in gene expression were examined by microarray analysis using rice roots at 3, 6, 9, 12, 24, and 36 h after the onset of Fe-deficiency treatment.
Indeed many of the genes reported to be regulated by metal deficiencies such as Zn deficiency changes in response to varying Fe availability (Ishimaru et al. [2012]; Suzuki et al. [2012]; Bashir et al. [2012]; Takahashi et al. [2012]).
There are conflicting reports on whether iron deficiency changes susceptibility to seizures.
In response to P deficiency, changes in ROS concentration and distribution in specific root cells occurred, although different patterns were reported (Potters et al., 2002; Shin et al., 2005; Tyburski et al., 2009).
Possible mechanisms linking cell wall and oil synthesis include increased sensitivity to stress caused by cell wall deficiency, changes in osmolarity or cell turgor, or changes in the nutrient (N) perception and sensing machinery or enzymes located inside the cell wall or in the periplasm [ 34].
Compared with wild-type islets, islets from Chop −/− mice also had reduced BiP mRNA expression, perhaps indicating that CHOP deficiency changes the balance between the induction of proapoptotic and prosurvival factors in the UPR pathway, resulting in reduced baseline ER stress in islets.
To determine whether increased levels of autophagy following olaparib and SAHA co-treatment is a direct result of PTEN deficiency, changes in the expressions of autophagy markers as well as autophagosome formation were assessed in MDA-MB-231 cells that were transfected with control or PTEN-specific siRNA.
Additionally we also characterized ob/ob, and Nscl-2 ×ob/ob compound mutant mice to study whether Nscl-2 deficiency changed the fat cell phenotype of the leptin deficient ob/ob strain.
DNA microarray analysis indicated that the Ecs deficiency changed expression of the virulence factor regulator protein Rot accompanied by differential expression of membrane transport proteins, particularly ABC transporters and phosphate-specific transport systems, protein A, adhesins and capsular polysaccharide biosynthesis proteins.
