Eyraud divides the collateral pathways into four systems: deep, portal, median, and superficial [18].
Specific IVC variations and anomalies including associated venous variations such as continuous azygos (CA) and hemiazygos CHA) veins and other collateral pathways (deep, portal, median, and superficial) were recorded using a standardized form.
In the first case, we encountered infrarenal agenesis and azygos continuation of IVC with active superficial, deep, and portal collateral pathways (Fig. 1).
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Persons without a functional spleen, with hereditary or chronic hemolytic anemia, and even without hematological conditions are at increased risk of atherothrombosis, deep vein, portal and superior mesenteric vein thrombosis, and pulmonary arterial hypertension [ 107, 110, 111], due to the prothrombotic state caused by higher thrombin formation [ 110].
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HIV, decongestive chronic liver disease, deep vein thrombosis, portal hypertension, jaundice and hemorrhoids.
These include, inter alia, deep vein thrombosis, portal vein thrombosis, Budd Chiari syndrome, nonbacterial thrombotic endocarditis, pulmonary thromboembolism, as well as disseminated intravascular coagulation [ 2].
Main outcome measures Adjusted rate ratios for all first time deep venous thrombosis, portal thrombosis, thrombosis of caval vein, thrombosis of renal vein, unspecified deep vein thrombosis, and pulmonary embolism during the study period.
The most frequent types of major thrombosis include stroke, transient ischemic attack, myocardial infarction, peripheral arterial thrombosis, deep venous thrombosis, portal vein thrombosis, and thrombosis of the hepatic veins causing Budd-Chiari syndrome [ 23, 24].
