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We found unilateral nigro-striatal dopaminergic loss to decrease swing speed of the contralesional forelimb and stride length of all paws in association with depletion severity.
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Furthermore, decreased swing speed, stride length and body speed correlated highly with lpTh hypometabolism.
CStr hypermetabolism was strongly associated with several pathological gait changes (decreased swing speed, stride length and body speed) but with only one compensatory effect (increased stance duration).
Compared to the control group, the two patient groups tended to walk more slowly, with decreased swing time % and increased stride time.
Induction of OA in transforming growth factor β1 injected mice by treadmill running also led to increased stance times and decreased swing time (15).
Compared to the control group, the two patient groups tended to walk more slowly and with decreased swing time and increased stride time.
For example, as average speed increases there is a small but noticeable decrease in swing phase duration in the forelegs when compared to other segments.
Increasing and decreasing pattern swing directions can be positive or negative at any time depending on current opening pattern swing properties.
Dopamine depletion significantly decreased CF swing speed in association with depletion severity, as supported by the hypokinesia and rigidity found in human PD patients [17].
The effect of the decreased clock swing on the local timing is analyzed: The degradation in the timing slack is shown to be insignificant due to bounded clock slew eliminating most of the timing degradation on the clock network or the logic paths induced by decreased clock swing.
By increasing the stretching parameter S will increase the skin friction (f 0)), while film thickness (beta=Upsilon^{frac{1}{2}}) decreases, but swing impact is detectable in both free temperature (theta(1)) and heat flux (-theta'(0)) when (K=0.1) as shown in Table 8.
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