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It was confirmed that SCF did not cause decrease in viability of the cells when they were exposed to SCF dosages below 10 μg/ml (Fig. 11a, b).
Minor decrease in viability of resistant cells treated with 5 μg ml−1 at 48 h was restored at 72 h (Fig. 6b).
The results indicate that exposure of Huh7 cells to TD triggers the pathway of apoptosis leading to decrease in viability of Huh7 cells.
The engagement of Fas receptors with simultaneous targeting of Bcl-2 resulted in a significant (P<0.0001) decrease in viability of clone nos.
Previous publications indicate that LPS alone would not cause cell death, but LPS combined with cytokines, i.g., interferon-γ (INF-γ) would cause decrease in viability of rat C6 and rat primary astrocytes [ 20, 21].
In contrast, monoHER protected NeRCaMs against doxorubicin-induced membrane damage (as illustrated by the increase in the LDH release from treated cells) and decrease in viability of treated cells.
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Decrease in viability and adhesion of B16F10 cells was accompanied by enhanced expression of caspase-3.
In comparison, cells co-treated with 50 μM ascorbic acid did not increase or decrease in the viability of the cells at all the tested concentrations.
Treatment with ODN CpG-2216, ODN CpG-2006 or ODN GpC-2006 resulted in similar respective decreases in viability of Akata cells versus Akata31 cells due to apoptosis.
After 4 h at a concentration of 100 μg/mL the following changes were observed: all CNTs produced significant decreases in viability of EAhy926 cells and only CNTs with diameters <8 nm induced significant decreases in L929 cells.
Both types of CNTs induced significant dose-dependent decreases in the viability of PC-12 cells, whereas graphene exerted adverse effects on the neural cells just at over 62.5 ppm.
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