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Effects of treatment with antipsychotics were not limited to decrease in elevated thyroid hormone concentrations.
In our patient cohort, rosuvastatin treatment resulted in a slight, non-significant decrease in elevated circulating vWF levels.
The decrease in elevated serum AST level might indicate the protective action of either CRAE or BB on the hepatocyte.
A decrease in elevated serum HER-2/neu levels to under 15 ng/ml or levels continuously below 15ng/mll during the course of disease correlated with significantly longer survival.
Treatment with cisplatin alone did not show any significant (P < 0.05) change in elevated serum insulin levels; however, treatment with combination of cisplatin with telmisartan produced significant (P < 0.05) decrease in elevated serum insulin levels in cancer treated rats).
Conversely, systemic (20, 26, 27) and intracerebral (28) bromocriptine administration in insulin-resistant animals leads to a decrease in elevated VMH noradrenergic and serotonergic levels (measured in vivo by microdialysis) with a resultant decline in hepatic glucose production/gluconeogenesis, reduced adipose tissue lipolysis, and improved insulin sensitivity.
Similar(54)
NRG1 was identified by microarray analysis to be decreased in cirrhosis, elevated in dysplasia, and again down-regulated during all four stages of HCC [ 4].
Elevated intermediate concentrations may be counteracted by a decrease in production and/or elevated consumption of these intermediates.
Principally, the need to decrease pathologically elevated glycemia in critically ill subjects has been generally accepted, although the exact target range in various patient subgroups is subject of ongoing discussion [ 1].
The observation that preproenkephalin is increased in the rVLM after repeated EA in the hypertensive rats, suggests that EA-associated increases in expression of preproenkephalin enhances the availability of rVLM enkephalin to, in turn, decrease elevated neuronal activity, sympathetic outflow and ultimately hypertension.
The mechanism of this disruption is linked, not to depletion of S-adenosylmethionine, as is often a feature of mammalian tumours, but to a decrease in choline and elevated S-adenosylhomocysteine, a potent inhibitor of DNA methytransferase.
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