Sentence examples for death were studied from inspiring English sources

In this experiment, the possible mechanisms involved in both blastomere proliferation and regulation of cell death were studied by analysis of relative expression patterns of IGF-II, BCL2-L1, Band, and HSP70 in 3 classes of morphological quality groups (e.g., excellent, good, and poor) of bovine blastocysts produced by IVF.

Two patterns of causes of death were studied: drug related death versus non drug related death.

The relationships of eight health and health behaviour characteristics with injury death were studied with adjusted Cox's proportional hazard model.

A number of other factors with potential to modulate cell death were studied, but none was regulated by cAMP or correlated with cAMP protection against DNR.

The variables sex, age and underlying, associated or total mentions of causes of death were studied using mortality rates, proportions and historical trends.

The effects of TF/FVIIa on cytokine-induced beta cell death were studied in MIN-6 cells and human pancreatic islets using cell-death ELISA and propidium iodide and cleaved caspase-3 staining.

However, causes and characteristics of patients' death are studied only in particular pathologies such as sepsis, cardiac arrest or ARDS ([1, 2]).

Caspase-3 activity, as executioner protease of neuronal death, was studied [17], [18].

Cell death was studied using a trypan blue staining as described by Mauch-Mani and Slusarenko [53].

Mesothelial cell death was studied in mice with S. aureus peritonitis and in mice injected with tumor necrosis factor alpha and interferon gamma.

Cell death was studied using TUNEL assay on cryosections (Flourescein Insitu Cell death detection kit, Millipore, Billerica, MA, USA).