Sentence examples for death were assessed from inspiring English sources

Epidemiological data, diagnosis at admission and estimation of risk of death, were assessed by APACHE II scores.

The mechanisms involved in TNF-α-induced cell death were assessed by using microarray analysis.

In two experiments, wild-type and ICOS−/− or wild-type and ICOSL−/− mice were treated with BrdU, and BrdU incorporation during treatment (due to proliferation) and BrdU loss after treatment (due to proliferation and to death) were assessed.

ROS production and cell death were assessed using flow cytometry.

Neurogenesis and cell death were assessed by immunocytochemical analysis.

Hazard ratios (HRs) and 95% CIs for cancer death were assessed by the Cox proportional hazard regression model.

Impact of transfusion on death was assessed among those subgroups.

Additionally, cause-specific death was assessed retrospectively, based on chart abstraction.

Cell death was assessed by LDH release assay and cell viability was assessed by CCK-8 assay.

The ability of this index to determine the primary end point (rehospitalization for HF or cardiac death) was assessed.

The association between PELOD-2 score and death was assessed by comparing the PELOD-2 score between survivors and non-survivors with a Mann–Whitney test.