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Caspase-3 activity, as executioner protease of neuronal death, was studied [17], [18].
Cell death was studied using a trypan blue staining as described by Mauch-Mani and Slusarenko [53].
Mesothelial cell death was studied in mice with S. aureus peritonitis and in mice injected with tumor necrosis factor alpha and interferon gamma.
Cell death was studied by detecting the TUNEL positive cells (TUNEL) and cell proliferation was detected by following the nuclei labeled with phospho-histone-3 (PH3), which identifies cells in the M phase of the cell cycle (Figure 5 A), or by BrdU incorporation (Figure 5 C).
The protective effect of D. reticulata extract against alloxan-induced cell death was studied by MTT assay.
The effect of CCR7 activation for MC survival in Fas/CD95-induced cell death was studied with three different methods.
Similar(48)
However, causes and characteristics of patients' death are studied only in particular pathologies such as sepsis, cardiac arrest or ARDS ([1, 2]).
Two patterns of causes of death were studied: drug related death versus non drug related death.
When BC-specific death is studied as the primary outcome, information on other causes of death such as cardiovascular (CV) disease is rarely addressed in the analysis [ 4].
A number of other factors with potential to modulate cell death were studied, but none was regulated by cAMP or correlated with cAMP protection against DNR.
The variables sex, age and underlying, associated or total mentions of causes of death were studied using mortality rates, proportions and historical trends.
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