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Cell death was evaluated by the TUNEL method.
The time to death was evaluated by a Cox proportional hazard regression analysis.
The photoprotective potential of this compound against UVA-induced oxidative damage and cell death was evaluated in cultured primary skin fibroblasts.
The average causal systemic antifungal therapy de-escalation effect on 28-day death was evaluated by using a double-robust inverse probability of treatment weight estimator which is a causal inference method based on observational data.
Cell death was evaluated by trypan blue dye exclusion and a more efficient specific cytotoxic effect on Caco-2 cells was observed on the cells incubated with the mixture of broccoli and green tea than on cells exposed to control infusions.
Severe morbidity, defined as hospital referral or death, was evaluated using Kaplan-Meier curves.
Neuronal death was evaluated at 5 hrs by combining phase contrast and fluorescent microscopy observations.
Cell death was evaluated at E9.5 using a Caspase 3 antibody and scored as above.
Apoptotic death was evaluated by anti-active caspase-3 staining (AC3)(R&D systems Minneapolis, USA).
Cell death was evaluated by cellular incorporation of propidium iodide (PI) 3d and 6d after Aβ treatment.
Association between risk factors and death was evaluated using Cox Regression Models adjusted by the intervention group.
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