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In multivariable-adjusted models including BMI, log-leptin squared was not related to CVD death (P = 0.70) but was associated with non-CVD death (P = 0.006).
Endotoxin exposure protected isolated cardiomyocytes against hypoxia-induced cell death (p < 0.001).
Black patients were 1.6 times more likely to die of a cardiac death (P <.001) than white patients.
Higher EMP2 expression was associated with a better progression-free survival (P = 0.008) and cancer-related death (P < 0.001).
Mortalities due to paratuberculosis were strongly determined by the level of exposure; sheep exposed to high doses had 18 fold higher odds of death (P = 0.007).
Cox's proportional hazard model for survival analysis showed that biopsy type had no significant effect on recurrence (P =.30) or melanoma-related death (P =.34).
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Cellular states are represented by four states, the proliferating (PR), pre-reproductive death (p-RD), reproductive death (RD), and pre-interphase death (p-ID) states, as shown in Fig. 5.
A test for trend was not statistically significant for either cause of death (P-values=0.40 and 0.07, respectively).
The topmost inhibited function was 'organismal death' (p-value 1.96E-03; 150 molecules; activation z-score -11.38).
HIV gp120 alone induces hepatocyte death (p<.002), as previously described [15].
24% died during admission: older age, time in hospital before bacteremia, and an unidentified infection focus were independent predictors of in-hospital death (p<0.001).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com