Sentence examples for death in normal from inspiring English sources

As they examined cell death in normal mouse ovaries, however, they began to realize that oocytes and follicles were dying so fast that a female mouse should exhaust virtually her entire natal complement of eggs by young adulthood.

Because Cr VI) is mutagenic and carcinogenic at doses that evoke cell toxicity, the objective of these experiments was to examine the effect of Cr VI) on the growth, survival, and mode of cell death in normal human lung fibroblasts (HLF cells).

These results agree with previous data reported that MDM2 antagonists induce reversible cell cycle arrest but not cell death in normal cells [8] and indicate that wild-type-p53 glioblastoma cells are more sensitive to nutlin-3a treatment than NHA.

Because of the antagonizing role p38 MAPK plays, we sought to better understand how p38 MAPK signals to promote cell survival and cell death in normal and chronic wounds.

The Antp-LP4 peptide did not induce cell death in normal peripheral blood lymphocytes (PBMCs).

Conversely, selenite failed to cause apoptotic death in normal blood cells (Supplementary Figure S1a).

The relatively new concept of apoptosis - programmed cell-death in normal tissue - has also led to a fundamental new comprehension of how treatments may work against tumours.

Every year chickenpox causes about 9,000 hospitalizations and results in about 50 deaths in normal children.

Some scholars have concluded that about 17 million people died, while other counts go as high as 45 million, reflecting varied assumptions about the death rate in normal times as well as other uncertainties, including how much official statistics undercounted deaths during the famine years.

However, ER-induced autophagy may be cell specific because it was seen that in colon and prostate cancer cell lines activation of autophagy by chemical inducers reduces cell death, whereas in normal human colon cells and in nontransformed embryonic fibroblasts, it contributes to cell-death [ 48].

In order to investigate the relative contribution of RIPK1 to inflammatory and cell death pathways in normal human macrophage development and function, in this study we have generated KO of RIPK1 in human iPS cells, and used a well-established model of iPS-MΦ differentiation.