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Multiple symptoms, signs, and clinical diagnostic data were common to the first 10 reported cases.
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Approximately 12,000 genes (depending on how many probes were discarded due to missing data) are common to both platforms.
An initial exploration of pathways affected shows that some of the pathways and transcription factors implicated by the transcriptional data are common to both mutants.
List of 211 differentially expressed genes from human array data that were common to the three mouse data sets.
Eleven data fields were common to both the UPMC electronic and the ACHD paper-based databases (Table 2).
This process revealed that certain data elements were common to all perspectives, while others were only needed for a more in-depth searching of users focusing on disease.
Discrepancies in pertussis-related data were common for up-to-date status (22.6%), number of immunizations (34.7%), dates (10.2%), and formulation (34.4%).
A set of DEPs and pathway network data were common and specific to each NFPA subtype.
For the density plots, we extracted the values for each data set that were common to all data sets.
Out of the 968 genes that could potentially overlap between these data sets, 634 genes were common to both (64.46% overlap, χ2 = 7458.18, df = 1, P<0.0000001).
We report data for the exposures that were common to both studies, 150 μg/m CAP+O3 and PFA.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com