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Heart dysfunction was detected only by serum troponin levels and the hemodynamic data: stroke work.
Similar to our data, stroke was not associated with OA in Ong et al's 27 study.
In the administrative data, stroke was the only condition defined exclusively from hospital records, while all other conditions were defined from both hospital records and physician billing claims.
Based on the retrieved patient-specific data, stroke subtypes were verified and further classified into cerebral infarcts, intracerebral hemorrhages, or SAHs.
We assessed burden of disease by reference to the following data: stroke subtype (ischaemic, hemorrhagic, TIA); history of stroke in the previous year; comorbidities (aphasia,hemiparesis, dysphagia, diabetes, chronic kidney insufficiency or heart failure) and geriatric multimorbidity.
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The following data were collected in this sample: NEI-VFQ (VRQoL) and SF-36 (HRQoL), demographic data, stroke-type (i.e. ischemic or hemorrhagic), visual field examinations, topography of the visual field loss (i.e. VFD-type), and visual acuity.
Hemodynamic datas, stroke volume (SV) and CO with two monitoring were performed at baseline 2 min before passive leg raising (PLR), 30 s after PLR and 2 min after volume expansion (VE) of 500 ml of saline solution.
Six secure studies provided data for stroke; β-blockers significantly increased stroke (RR 1.73, 95% CI 1.00 to 2.99, p=0.05).
Because data for stroke subtypes from south Asian cohorts were limited, separate analyses for ischaemic stroke and haemorrhagic stroke were conducted for east Asians only.
We collated the data of stroke patients who underwent thrombolysis through the SITS-ISTR (Safe Implementation of Treatment in Stroke-International Stroke Thrombolysis Registry, www.sitsinternational.org, held at Karolinska University Hospital, Stockholm) from December 2002 to November 2009.
Fourth, we lack data regarding stroke subtypes thus we were unable to examine the association between SED and mortality in patients with haemorrhage stroke.
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