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* p = 0.30 ** p = 0.20 † p = 0.053 &p = 0.69 All Chi-square analyses with not recorded data excluded in the calculation.
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Another explanation invokes the failure to include incipient species as distinct units in our data, excluding in such way from the analyses recent cladogenetic events [ 70, 71].
Since in vitro data excluded ischemic S100A1 production in CFs, our results support the view that S100A1 is passively released from damaged cardiomyocytes and subsequently internalized by adjacent CFs.
It is important to note that our previous pathogenic classification for G1738R was not driven by immunohistopathology results but rather by the observation of loss of the wild-type allele in both BRCA1 G1738R tumours studied, data we excluded in this study due to the observation that our loss of heterozygosity data on classified variants do not support the underlying assumptions of the model.
If the outlier's data are excluded in the analysis, this relationship is even smaller, r(24) = –0.06, p = 0.77.
It will decrease the probability of the most frequently occurring negative temperature gradient if data are excluded in this time zone.
One study without detailed genotyping data was excluded in the analyses [24].
New Zealand data are excluded in this analysis.
Those data were excluded in this study.
Gaps and missing data were excluded in the calculations.
Missing data were excluded in the logistic regression analysis.
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