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Our data demonstrated that each family has a particular amplification pattern, with 7 families having copies recently inserted within the last 0.2 million year.
The binding stoichiometry revealed by the complementary mass spectrometry data demonstrated that each apo-rMT II molecule could bind up to six iAsIII, 10 MMAIII, and 20 DMAIII, consistent with the coordinate chemistry of the individual trivalent arsenicals.
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These data demonstrate that each muscle may have a different scaling relationship between single-fiber and fiber-bundle levels, suggesting that the structures responsible for higher order passive mechanical properties may be muscle specific.
The data demonstrate that each revertant strain possesses wild-type levels of CENP-ACnp1.
Together, these data demonstrate that each germ layer is associated with a specific level/pattern of cyclin D expression.
The data demonstrate that each MEC population is able to generate tumors at similar latency and with broad pathology.
These combined data demonstrate that each of the changes in the B-cell compartment in WASp−/− mice reflects a B cell intrinsic defect.
Together these data demonstrate that each GPN family member plays a role in the localization of RNAPII subunits and that soluble subunit levels are also altered either because of instability or reduced expression.
As predicted, our data demonstrated that in each of the six brain reward ROIs, viewing images of food was associated with a greater BOLD fMRI signal than viewing nonfood images in the fasted state.
Nevertheless, our data demonstrated that bivalent CD4 binding was not sufficient to induce CD4 down-modulation.
Similarly, data demonstrated that smoking was a major killer decades before the biological processes were understood.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com