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These models contradicted the data demonstrated by Smith and Vinograd [24] that in cycloheximide-arrested human cells eccDNA consists of pre-existing DNA.
Human AG O DNAinteraction data demonstrated by ChIP-seq experiment were obtained from the gene expression omnibus (GEO), GSE40536 experiment of Ago1 ChIP-seq (GSM995943) published by Huang et al.
One surprising aspect of these data demonstrated by New et al. (2006) was that a U-shaped pattern of word-length effects became apparent when they tested the performance of lexical decision across the full ELP dataset of over 33,006 words.
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Our data demonstrate, by several independent methods, an F508-dependent interaction between CFTR and IgG.
Together, these data demonstrate by multiple measures that all mutations are not functionally equivalent and can have distinct effects on collagen expression and assembly.
Therefore, our experiments on the synthetic data demonstrated that, by considering all three types of identity attributes and matching references in a collective and iterative manner, CR is a more effective method for identity resolution.
Our previously published data demonstrated that by depletion of the pDC subset we were able to significantly alter the immune response to RSV in vivo [14].
Our data demonstrated that by targeting to PDK1 and blocking its kinase activity, NA potently inhibited the PI3-K/Akt pathway and its downstream metabolic regulator HK2, which eventually resulted in ATP depletion and dysfunction of glucose metabolism.
Our initial data demonstrated channel capture by C terminal fusion proteins from brain crude membrane lystate.
These data demonstrated that autophagy activation by Rapa reduced cell apoptosis by PA treatment.
These data demonstrated that autophagy inhibition by CQ promoted PA-induced apoptosis in hepatocytes.
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