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Similarity in tissue distribution data and bone uptake levels for the two groups of mice indicate that the defluorination might be resulting from weaker C F alkyl bond and perhaps not due to tumor mediated catabolic defluorination of the parent molecule.
The demographic data and bone density parameters for the study subjects are detailed in Table 1.
The Kruskal-Wallis test [ 17] was used to evaluate non-parametric haemograms, biochemical data and bone marrow aspirate results.
A 3D model of the allogenic bone was prepared from CT scan data, and bone defect reconstruction and the outcome of bone defect repair were simulated before surgery.
Differences in cartilage invasion score, histologic data, and bone erosion score between the treatments groups were assessed by Kruskal Wallis test followed by the Mann–Whitney U test.
This KNLR provides a tool for measuring the bone turnover that correlates with biochemical data and bone histomorphometric analyses performed on biopsy samples [ 14, 15].
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Study design: Adolescent girls with AN (n = 61) had anthropometric, nutrition, and exercise data acquired, and bone mineral density (BMD) and body composition measured by dual energy x-ray absorptiometry.
One hundred ninety five came for an initial study visit and had baseline demographic data collected and bone densitometry done, but 19 were judged at that point not to meet the study inclusion criteria.
To test the representation of individual species' abundances between the death assemblage and the living community, 95% bootstrapped confidence intervals were calculated [59] for each species' proportional abundance and compared between the living (survey data) and dead (bone MNI) representations of that species.
Statistical significance of the biomechanical data and the bone volume data was determined by using the Mann–Whitney U-test.
Our primary blood Pb analyses included 873-918 withn with complete data and our bone Pb analyses included 568-594 withn with complete data.
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