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Oxidative damage was determined by quantifying nitrotyrosine content.
The DNA damage was determined by agarose gel electrophoresis.
Cells were then harvested and DNA damage was determined with the Comet assay (Chen et al. 2009b).
The extent of histologic lung damage was determined using quantitative stereological techniques as previously described [22, 24].
In this FE-based approach, impact damage was determined by maximum-stress criteria in several failure modes.
The maximum depth of the exfoliation damage was determined by an ultrasonic non-destructive inspection (NDI).
The stages of apoptosis were evaluated by AO/EB staining and nuclear damage was determined by Hoechst 33258 staining.
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The extent of damage is determined by the anatomic distribution of coronary blood flow.
Damage is determined according to damage index based on axial load-bearing capacity.
Chloride diffusion coefficients of concrete undergoing fatigue damage were determined by a modified RCM method.
After impact the area of damage is determined using ultrasonic techniques.
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