Sentence examples for damage the expressions from inspiring English sources

The linear undamped modal eigenproperties are then extracted, in closed-form, from the linearized equation of motion, thus generalizing to the presence of an arbitrary damage the expressions known from the literature for undamaged suspension bridges.

After UV-induced damage, the expression of genes involved in DNA repair was silenced universally;30 conversely, many miRNAs were upregulated by UVB (280 320 nm) irradiation.31,32 In this study, OTUB1 (OTU deubiquitinase, ubiquitin aldehyde binding 1) was expressed following UVB irradiation, causing p53 stabilization, which was not downregulated even by lethal UVB irradiation33 (Figure 3).

Although endothelial cells from glomerular capillaries might drop into the urine because of glomerular epithelium (podocyte) damage, the expression of podocyte marker was not detected in Vwf-positive urine.

In the CNS, MA has been shown to increase excitotoxicity, BBB damage, the expression of proinflammatory cytokines, and oxidative stress.

To assess whether resveratrol causes DNA damage, the expression of phosphorylated H2AX and activated ATM in CD19+/CD5+ cells was measured.

In the absence of damage, the expression of both antioxidant and prooxidant genes showed no significant difference between Bmi1−/− and WT neonatal mice.

In response to persistent DNA damage, the expression of p53, p21 and p16 increased, ultimately contributing to halting cell proliferation to allow DNA repair to occur.

In response to DNA damage, the expression of MICA and MICB can be induced by ATM and ATR, which are components of DNA damage signaling pathways [ 24, 29, 30]; these effects can be prevented by ATM/ATR inhibitors.

To investigate whether cells displaying unrepaired thymine dimers were able to enter S phase following UV-induced damage, the expression of the cell cycle progression marker cyclin A in these cells was assessed by immunocytochemical staining.

Specialized compounds are not continuously expressed, but may be produced as a response to herbivory or other damage, the expression may also be dependent on the environment [ 38] and plants often use a combination of several defensive traits [ 7, 17].

Whilst the RPE/Bruch's membrane may be the primary site of complement-mediated cell damage, the expression of complement regulatory proteins in the neural retina [ 6] suggests that in disease, photoreceptors may themselves become vulnerable to direct assault from C3 and its breakdown products or C5b-9.