Sentence examples for damage stabilization from inspiring English sources

Presented overview of experimental study of lithium CPS in plasma devices demonstrates the progress in protection of tokamak plasma facing components (PFC) from damage, stabilization and self-renewal of liquid lithium surface, elimination of plasma pollution and lithium accumulation in tokamak chamber.

These findings suggest that drought tolerance of chrysanthemum plants may be related to the regulation of hormone biosynthesis and signaling, reduction of oxidative damage, stabilization of cell proteins and structures, and maintenance of energy and carbon supply.

Femoral and tibial nailing with reaming is used commonly after damage control stabilization with external fixation for cases of multiple trauma [16] or open fractures of Gustilo type III [49, 50].

In contrast, in CDV-exposed malignant cells, as a consequence of DNA damage accumulation, stabilization of p53 and induction of several pro-apoptotic genes (such as BIK, CYLD, DKK3, PLAU, and TIMP3) take place.

DOI: http://dx.doi.org/10.7554/eLife.04883.013 To determine the contribution of Hausp to the damage-induced stabilization of nuclear Cry1, we examined nuclear Cry1 protein levels following DNA damage in MEFs expressing either control sequences or Hausp-targeting shRNA.

Indeed, depletion of Hausp prevents damage-induced stabilization of nuclear Cry1, similar to the effect of Hausp depletion on p53 accumulation.

The use of siRNA to CHK1 or CHK2 does not abrogate the damage-induced stabilization of p53 [ 43], and the knockout of CHK2 in cancer cell lines does not compromise Ser20 site phosphorylation [ 44].

The number of sites associated with each COG (Fig. 1D) reveals that the composite gut microbiome SOS regulon is dominated by four molecular functions: transcriptional repression (lexA), sensing of DNA-damage and stabilization of DNA (recA), translesion synthesis (umuD, dinB, uvrX and imuB) and excision repair (uvrB, uvrD and uvrA).

An interesting interaction occurs between NO and p53 tumor suppressor, in which NO-induced DNA damage causes the stabilization and accumulation of p53, which in turn, transrepresses inducible nitric oxide synthase (NOS2) in a negative feedback loop.

Activated DNA damage signalling caused stabilization of the tumour suppressor p53, which resulted in cell cycle arrest and apoptosis.

Because Cry1 and Cry2 each can repress the other's expression, Cry2 protein could decrease in response to damage secondary to stabilization of Cry1.