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Compared to leg length discrepancies, arm length discrepancies are less frequent and subsequent secondary damage is lower [2].
The level of damage sustained by cells determines whether programmed cell death (apoptosis) can unfold or, if the damage is lower, senescence can occur [ 150, 151].
The second conclusion is that the capacity to repair stress-induced DNA damage is lower in mitochondria than the nucleus, because mitochondria are the principal site of ROS production, employ fewer repair processes than do nuclei, or lack protective histones on their mtDNA molecules [ 17, 18, 27, 29- 31].
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For both uprooting methods, dug losses were slightly higher and superficial beet damage was lower compared to share lifting.
Leaf skeletonization was negatively correlated with elevation and leaf miner damage was lower in high density plant populations.
Low-salinity water injection precipitated calcium sulfate because of the low solubility of calcium sulfate in low-salinity water, but the damage was lower than that caused by high-salinity seawater.
Fields with higher proportions of orchard grass showed higher levels of grazing damage; and damage was lower in years when abundant alternative feeding areas (AFAs) in the form of unharvested potatoes were available.
Tolerance of leaf damage was lower in the low light environment.
The odds of developing target organ damage was lower in women compared to men in all four models (Table 3).
The odds of developing hypertensive target organ damage was lower in women compared to men and this was partly explained by the current BP level.
Interestingly, DNA damage was lower at 25 µM TQ in normal fibroblast cells (IMR90 and hTERT-BJ1) than in glioblastoma cells.
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CEO of Professional Science Editing for Scientists @ prosciediting.com