Sentence examples for cyclic elevation from inspiring English sources

Although findings are conflicting and primarily relate to responses of individual cells (excluding the cross-talk between cells), some evidence indicates that short-term cyclic elevation in pressure induces anti-atherogenic changes in the arterial wall (for a review, see [ 97]).

Our main findings were that lungs exposed to cyclic elevations in pulmonary artery pressure in the absence of ventilation formed less edema and exhibited less perivascular and alveolar hemorrhage than did ventilated lungs exposed to similar peak and mean pulmonary artery pressures and mean airway pressure [ 26].

The results from these studies demonstrated that all methods targeting cyclic AMP elevation were able to enhance regeneration, though forskolin and 8-bromo cyclic AMP had the most profound effect on axonal elongation, and theophylline produced the largest decrease in the initiation time required for neurite sprouting.

Although cyclic strain caused an elevation in proteoglycan expression regardless of cell type, the type of proteoglycan differed between groups: the Dcn−/− cell-seeded gels produced an excess of biglycan not found in the wild-type controls.

Our findings show that inhibitory effects of fMLP-induced human eosinophil chemotaxis by BAY 41-2272 at short-term or prolonged exposition time are accompanied by significant elevations of cGMP and cAMP, but we could not detect a clear correlation between chemotaxis inhibition and elevation of cyclic nucleotide levels.

Earlier we reported that maggot secretions inhibited the pro-inflammatory responses of human neutrophils [14] and monocytes [15] through elevation of cyclic AMP.

Activation of RXFP1 by relaxin caused elevation of cyclic adenosine monophosphate (cAMP) levels [ 15, 16].

Endogenous platelet inhibition is predominantly based on cyclic nucleotides (cAMP, cGMP) elevation and subsequent cyclic nucleotide-dependent protein kinase (PKA, PKG) activation.

Free radicals, proinflammatory cytokines, metabolic acidosis, electrolyte disturbances and elevation of cyclic guanosine monophosphate (cGMP) have been referred to as possible causative factors [ 1– 3].

This effect was transmitted via the BNP receptor guanylyl cyclase A (GC-A), elevation of cyclic guanosine monophosphate (cGMP) and activation of protein kinase G (PKG) [ 17].

Nitric oxide seems to exert its action on muscle glucose transport partly via activation of guanylate cyclase, leading to elevation of cyclic GMP (cGMP) levels [ 10, 12].