Sentence examples for cycle exposure from inspiring English sources

Primary environmental variables include light, heat and moisture; procedural variables include exposure cycle, exposure time and test initiation.

The results showed for the first time, a life cycle exposure to MC-LR caused growth inhibition, decreased ovary weight and ovarian ultra-pathological lesions.

The results, for the first time, show a life cycle exposure to MC-LR causes growth inhibition, testicular damage and delayed sperm maturation.

Azcaxalli's objective function involves thermal limits at the end of the cycle, cold shutdown margin at the beginning of the cycle and the neutron effective multiplication factor for a given cycle exposure.

Our findings indicated that a life cycle exposure to MC-LR causes endocrine disruption with organic and functional damage of the testis, which might compromise the quality of life for the survivors and pose a potent threat on fish reproduction and thus population dynamics in MCs-contaminated aquatic environments.

Our findings indicate that a life cycle exposure to MC-LR impairs the development and reproduction of female zebrafish by disrupting the transcription of related HPGL-axis genes, suggesting that MC-LR has potential adverse effects on fish reproduction and thus population dynamics in MCs-contaminated aquatic environment.

Life-cycle exposure to MC-LR caused delayed ovarian maturation and sperm development along with ultrapathological lesions in the brain and liver.

The purpose of this study was to characterize the thyroid endocrine disruption induced by life-cycle exposure to BDE-47 in adults and offspring of zebrafish (Danio rerio).

Here we use DEB theory and explicitly a recently developed version of the DEBtox model [ 17, 19], to investigate the physiological mode of action of three different types of chemical stressors (the metal cadmium, the non-polar organic fluoranthene, and the herbicide atrazine) during a whole life-cycle exposure of the nematode Caenorhabditis elegans.

In the F1 generation (after full life-cycle exposure, 314 dpf), 284 adults from six replicate tanks in each of the remaining exposure groups (0.5 ng/L and 5 ng/L EE2 treatments with no F1 exposure; 0.5 and 5 ng/L EE2 and 5 ng/L E2 treatments with exposure stopped at 75 dpf in F 1 ) were weighed; we then collected blood samples and determined the hematocrit value.

To study the effect of repetitive hypoxia/reoxygenation on breast cancer cells, we optimized conditions of hypoxia and reoxygenation with regard to the number of cycles, exposure time, cell numbers, and other parameters.