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Absence of a cuff leads to superior escape of the humeral head, eccentric positioning of the humerus within the glenoid and poor glenohumeral control.
Codman [2] hypothesised that overuse degeneration of rotator cuff leads to calcific deposits in the tendon, and this was also supported by Bishop [12], whereas Sandstrom [13] proposed that the degeneration in the tendon follows local ischaemia which led to calcium deposition.
In clinical practice, abrupt loading to the repaired rotator cuff leads to a reduction in the tensile strength of the repair [ 1].
It is, however, also the case that an inflated exsanguination cuff leads to blood pooling [ 3] at the height of and distal to the affected section of the limb.
It has become apparent, however, that pooling and leakage around the endotracheal tube cuff leads to aspiration of contaminated oropharyngeal secretions, stomach contents and bacteria into the trachea and the lower respiratory tract [ 16].
The fully inflated cuff leads to limited or even absence of channel formation and the shape of the cuff results in a larger surface area in contact with the trachea.
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proximal cuff led to secondary distal reimplantation.
Irritation and pain caused by the cord, penetrating the rotator cuff, led to its removal in patient Y.G.
The progression of a complete cuff tear leads to rotator cuff arthropathy (osteoarthritis).
Fixed angular stability and meticulous rotator cuff repair leads to early mobilization and satisfactory functional outcome.
Poor biomechanics, especially with fatigue of scapular stabilizing and rotator cuff muscles, leads to dynamic instability and impingement [19].
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