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A more likely explanation is that the discrepancy stems from a time difference between rat and human in how the phosphorprotein activates downstream genes.
A possible explanation could be that neurons need signal from the astrocytes to respond effectively.
There are several possible explanations, stemming from different viewpoints, as to why DAG accumulation leads to IR.
A possible explanation for this difference could be that cultured fibroblasts obtained from fibrotic lesions have lost specific characteristics.
A possible explanation could be that particles from different locations have different effects on (DP_{loss}).
One likely explanation could be that women are more likely to suffer from conditions that are debilitating but not fatal.
A historical explanation could be that short introns were prevalent before ciliates diverged from other eukaryotes.
A contributory explanation could be that detrimental effects of gender inequality on mental health emerge from accumulation over time [ 46].
The hope is that ultimately stem cells from adults could be "reprogrammed" to cure diseases.
One explanation could be that TGFβ disruption impairs differentiation of LPCs after their activation, thus promoting their transformation into liver cancer stem cells [ 131].
One possible explanation could be that ubp1 regulates MDR1 ubiquitination.
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