These defects could, however, result from various initial defects including an alteration of auxin content, an alteration of auxin transport or a shift in auxin sensitivity.
The increase in condensin binding could, however, result from the delay in anaphase onset caused by Sgo1 overexpression.
The low LOD of this electrode could, however, result not only from the type of IL but also from application of Ni/Al hydroxide layers.
This staining, however, could result from the internalization of SCF after its ligation to KIT.
The phenotype of the nBmp2NLStm mouse, however, could also result from the dysfunction of several other proteins, because muscle contraction/relaxation cycles are quite complex.
The reduction of sister centromere splitting observed in the Sgo1 and condensin mutants could, however, equally well result from an increase in centromeric sister chromatid cohesion.
However, prosecution could result from "very excessive and gratuitous force", such as attacking someone who is unconscious.
However, they could result from the misuse of human regulatory regions in the mouse environment, or the omission of minor regulatory regions from the BAC construct.
In mice, the removal of Sox1 causes only microphthalmia (Nishiguchi et al., 1998); however, this could result from compensation by other members of SoxB1 gene family.
However, it could also result from some heating mechanism and/or an additional electron population.
However, it could equally result from dephosphorylation (e.g., by a serine phosphatase in the example of Figure 3A) of pre-existing doubly phosphorylated peptide (in blue).
