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One of the possible hypotheses to explain the potential mechanism underlying this interaction could be alteration in the oxidative stress-mediated inflammatory pathways in periodontal disease and PLBW [ 6, 26].
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One possible reason for this peculiar finding could be alterations of cell properties in the cortical tissue surrounding epileptic foci.
While there was no change in PTP1B expression, there still could be alterations in phosphatase activity and further studies are underway to explore this possibility.
One possible reason for this problem could be alterations in signalling pathways that are downstream or parallel to ErbB-2 upon trastuzumab or lapatinib treatment.
Previously it has been suggested that a possible explanation for the increased risk of autism among children whose parents have immigrated from Southern to Northern latitudes shown in some studies could be alterations in the immune repertoire due to differences in early pathogen exposure in mothers or neonates [ 29].
A consequence of blocking CGRP could thus be alterations in wound healing and increased inflammatory responses in skin injuries at the site of injection for the antibodies.
The mechanism could be an alteration of cellular cytokine production at the TKA site, given that the mean JIL-6 level was approximately 7 8 times the mean SIL-6 level.
The net result could be either alteration of calcium or redox signaling.
A part of the explanation of the observed effects of Pi and Zn nutrition on the growth capacity of both lettuce varieties could be an alteration in photosynthesis.
Another confounding factor that might interfere with alveolar derecruitment measured with the P V curve method could be an alteration of the chest wall elastance.
Because we obtained these results from whole lung homogenate, one explanation could be an alteration in the number and percentage of immune cell populations in the lung.
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