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starting point for future studies of cortical map development.
Cellular mechanisms for cortical map plasticity and stability.
Kilgard, M. P. & Merzenich, M. M. Cortical map reorganization enabled by nucleus basalis activity.
The cortical map used most frequently today by both researchers and clinicians is essentially the same one developed more than 100 years ago by anatomist Korbinian Brodmann.
These results are consistent with earlier observations that sensory input statistics can shape cortical map organization and spike timing.
These data suggest that transient SERT expression in specific glutamatergic neurons provides area-specific instructions for cortical map patterning.
Profiling mouse and human cortical transcriptomes have revealed temporal-specific differential gene expression modules in distinct neocortical areas during cortical map establishment.
However, the cortical map is distorted compared with the retina, with a disproportionately large area devoted to the fovea and its immediate vicinity.
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We use concepts from continuum mechanics to describe the retino-cortical map [36].
Even more importantly, Eq. 2 fails to describe the retino-cortical map close to the representation of the fovea ( θ < 1 ) because the foveal point at θ = 0 is a singularity.
Since dopamine [ 8] and acetylcholine [ 9] may influence cortical-map plasticity, an imbalance between dopaminergic and cholinergic systems, as assumed in schizophrenia [ 10], was discussed to account for the arbitrary spreading of activation and the formation of disordered associative networks.
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